ironjustice

Your 'smack on the head' .. in probability .. allowed hemosiderin /
iron from blood spilled to deposit in your ear .. and this iron
oxidizes and this .. or oxidation in particular has been shown to be
'involved' in destruction of hearing .. and it seems the TARGETING of
this oxidation is now being touted as a TREATMENT / reversal of hearing
loss ..

Neurobiology of Aging
Volume 27, Issue 7 , July 2006, Pages 1035-1044
doi:10.1016/j.neurobiolaging.2005.05.003
Copyright © 2005 Published by Elsevier Inc.
The effects of antioxidants in the senescent auditory cortex

C. de Riveraa, B. Shukitt-Haleb, J.A. Josephb and J.R. Mendelsona, ,


aDepartment of Speech-Language Pathology, University of Toronto,
Toronto, Ont., Canada M5G 1V7
bUnited States Department of Agriculture Human Nutrition Research
Center on Aging at Tufts, Boston, MA 02111, USA


Received 10 January 2005; revised 19 April 2005; accepted 2 May 2005.

Available online 13 June 2005.


Abstract
We investigated whether a 2-month dietary supplementation of
antioxidants, in the form of blueberry phytochemicals, could reverse or

retard the age-related decline in temporal processing speed observed in

the aged rat. To this end, extracellular single unit responses to
frequency modulated (FM) sweeps were recorded in the primary auditory
cortex (AI) of aged rats that had been placed on either a
blueberry-supplemented or control diet 2 months prior to the
physiological recordings. Results showed that most cells recorded from
the blueberry-fed rats responded most vigorously to fast FM sweeps,
similar to that observed in young rats. In contrast, the majority of
cells recorded from the control rats showed a preference for slow FM
sweep rates. These results suggest that age-related changes in temporal

processing speed in A1 may be reversed by dietary supplementation of
blueberry phytochemicals.


Keywords: Aging; Auditory cortex; Antioxidants; Temporal processing
speed; Frequency modulated sweeps; Blueberries


Corresponding author. Present address: Toronto Rehabilitation
Institute, University Centre, 550 University Ave., Toronto, Ont.,
Canada M5G 2A2. Tel.: +1 416 597 3422x3852.


Neurobiology of Aging
Volume 27, Issue 7 , July 2006, Pages 1035-1044

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J Laryngol Otol. 1997 Jan;111(1):60-2. Related Articles, Links


Superficial siderosis of the central nervous system: an underestimated
cause of hearing loss.


Castelli ML, Husband A.


Department of Otorhinolaryngology, Queen Alexandra Hospital, Cosham,
Portsmouth, UK.


Superficial siderosis of the central nervous system (CNS) is a rare
disease resulting in the ac***ulation of haemosiderin in the meninges,
the brain surface, the spinal cord and the cranial nerves. The pigment
is deposited as a result of chronic bleeding in the subarachnoid space.

This produces a clinical picture of deafness, ataxia, cranial nerve
deficits and in the latest stages dementia. In some cases the source of

bleeding can be identified, whilst in others it can not. Despite its
rarity the disease should be considered in the differential diagnosis
of sensorineural deafness, particularly as it is a progressive and in
some cases curable disease which is easily diagnosed by magnetic
resonance imaging (MRI). In this case report the haemosiderin was
derived from an ependymoma of the fourth ventricle with extension into
the cerebello-pontine angle. The first symptom was a worsening
sensorineural hearing loss.


Publication Types:
Case Reports


PMID: 9292135 [PubMed - indexed for MEDLINE]
---------------------------------------------------------------------------*-----
<<snip>>
This result confirm the vitamin
E to be an important factor of the normal process of nerve regeneration
<<snip>>

: Arch Ital Anat Embriol 1990 Apr-Jun;95(2):155-65 Related Articles,
Books,
LinkOut


Effect of vitamin E-deficiency on regeneration of the sciatic nerve.


Cecchini T, Cuppini R, Ciaroni S, Del Grande P.


Istituto di Scienze Morfologiche, Universita di Urbino, Italy.


The regeneration of the sciatic nerve fibres was studied in both normal

and
vitamin E-deficient rats at 30 and 60 days after crush. The vitamin E
is
involved in one of the most important mechanisms of protection against
peroxidation of plasma membrane lipids; the plasma membrane plays
certainly a
role in nerve regeneration. Both the diameter and the total number of
myelinated nerve fibres was calculated at different times. The number
of
myelinated fibres in the undenervated deficient animals was lower than
that
found in the undenervated normals animals. Following the nerve crush,
in normal
animals after two months the number of myelinated fibres exceeded the
number
found in undenervated normal animals, whereas in the deficient rat
nerves it
was significantly lower than in the corresponding controls and moreover

it did
not even reach the number found in the nerves of undenervated deficient

rats.
Finally, the caliber distribution of myelinated fibres in undenervated
and
denervated deficient rats shows a relative percent increase in the
number of
greatest axons and a decrease in smaller axons. This result confirm the

vitamin
E to be an important factor of the normal process of nerve
regeneration.


PMID: 2078094 [PubMed - indexed for MEDLINE]


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